@article {14077,
	title = {Role of transposable elements in trypanosomatids},
	journal = {Microbes and Infection},
	volume = {10},
	year = {2008},
	month = {2008/05//},
	pages = {575 - 581},
	abstract = {Transposable elements constitute 2-5\% of the genome content in trypanosomatid parasites. Some of them are involved in critical cellular functions, such as the regulation of gene expression in Leishmania spp. In this review, we highlight the remarkable role extinct transposable elements can play as the source of potential new functions.},
	keywords = {Cellular function, Domestication, Evolution, Gene expression, Leishmania, Regulation of mRNA stability, Retroposon, Transposable element, Trypanosoma},
	isbn = {1286-4579},
	doi = {16/j.micinf.2008.02.009},
	url = {http://www.sciencedirect.com/science/article/pii/S1286457908000464},
	author = {Bringaud,Fr{\'e}d{\'e}ric and Ghedin,Elodie and El-Sayed, Najib M. and Papadopoulou,Barbara}
}
@article {20053,
	title = {Genetic nomenclature for Trypanosoma and Leishmania.},
	journal = {Mol Biochem Parasitol},
	volume = {97},
	year = {1998},
	month = {1998 Nov 30},
	pages = {221-4},
	keywords = {Animals, Leishmania, Terminology as Topic, Trypanosoma},
	issn = {0166-6851},
	author = {Clayton, C and Adams, M and Almeida, R and Baltz, T and Barrett, M and Bastien, P and Belli, S and Beverley, S and Biteau, N and Blackwell, J and Blaineau, C and Boshart, M and Bringaud, F and Cross, G and Cruz, A and Degrave, W and Donelson, J and El-Sayed, N and Fu, G and Ersfeld, K and Gibson, W and Gull, K and Ivens, A and Kelly, J and Vanhamme, L}
}
@article {14084,
	title = {Multiple mechanisms of immune evasion by African trypanosomes},
	journal = {Molecular and Biochemical Parasitology},
	volume = {91},
	year = {1998},
	month = {1998/03/01/},
	pages = {51 - 66},
	abstract = {During infection of a mammalian host, African trypanosomes are in constant contact with the host{\textquoteright}s immune system. These protozoan parasites are infamous for their ability to evade the immune responses by periodically switching their major variant surface glycoprotein (VSG), a phenomenon called antigenic variation. Antigenic variation, however, is likely to be only one of several mechanisms enabling these organisms to thrive in the face of the immune defenses. The ability to grow in high levels of interferon-gamma (IFN-[gamma]) and to avoid complement-mediated destruction may also facilitate the parasite{\textquoteright}s survival. In this review we summarize (i) the activation of trypanosome genes for three different VSGs during antigenic variation, (ii) the secretion of a trypanosome protein that induces host CD8+ T cells to produce IFN-[gamma], and (iii) the evidence for trypansome protein similar to a surface protease of Leishmania that plays a role in resistance to complement-mediated lysis.},
	keywords = {Leishmania, Recombinant cloning, T cell, Trypanosomes, VSG genes},
	isbn = {0166-6851},
	doi = {16/S0166-6851(97)00209-0},
	url = {http://www.sciencedirect.com/science/article/pii/S0166685197002090},
	author = {Donelson,John E. and Hill,Kent L. and El-Sayed, Najib M.}
}
